17 Anthony Caiafa1 and Louis Visser2 1 School of Veterinary and Biomedical Sciences, James Cook University, Townsville, Queensland, Australia 2 Arizona Veterinary Dental Specialists, Scottsdale, AZ, USA Operative dentistry is that field of dentistry concerned with restoring defective areas of vital and non‐vital teeth back to normal masticatory function, periodontal health, and esthetics. Defects can be due to disease, infection, trauma, or abnormal development. Operative dentistry includes the application of material and instrument science in the treatment planning and restoration of the teeth. Many areas and concepts of restorative and prosthodontic therapy overlap (see Chapter 18 – Crowns and Prosthodontics), with other basic terminology covered in Chapter 1 (Oral Anatomy and Physiology). Specific terms relevant to restoration are covered here: When describing a cavity or restoration, the location can be described by the surfaces of the tooth that are involved. These are as follows: In order to properly treat lesions, they are best classified as to the tooth involved, type, and extent of lesion. Individual tooth identification systems can be found in the chapter on oral examination (see Chapter 2 – Oral Examination and Diagnosis). The following are some of the more common tooth pathology classifications dealing with type and extent. Classification by location includes the G.V. Black Modified Cavity Preparation Classification system (Table 17.1 and Figure 17.1), the Elementary Cavity Class, and the Practical Cavity Classifications. Table 17.1 G.V. Black modified cavity preparation classification system. Note: There may also be two separate lesions present on the same tooth (i.e., Class II, Class V) or a combination lesion where two locations are present and contiguous (i.e., Class II/V). Practical Cavity Classification [3] (Note that descriptions with “facial” are more appropriate for human dentistry): The American Veterinary Dental College (AVDC) Nomenclature classification of dental fractures can be found in Chapter 6 (Traumatic Dentoalveolar Injuries – TDI), along with discussion of expanding these descriptions to include root fractures, luxation, and alveolar injuries [4]. A previously used staging classification is listed below, but it is recognized that a more complete nomenclature listing may be needed. This more inclusive system would identify fractures into the enamel (Stages 1 and 8 below), enamel–dentin fracture (Stages 2 and 8), enamel‐dentin‐pulp fracture (Stages 3, 4, and 9), root fractured and displacement/avulsion (see Chapter 6 – TDI). Additional classification of tooth resorption (TR) can be found in Chapter 20 – Domestic Feline Oral and Dental Diseases. Generally, these TR lesions are not restored. These are generally referred to as stages and are used in combination with Black’s Modified Classification of tooth lesion locations (classification by extent of pathology). Stage: See Table 17.2. Table 17.2 AVDC abbreviations – restorative (https://www.avdc.org/traineeinfo.html; accessed 25 October 2017). In human dentistry, dentinal and pulpal inflammation and pain responses are considered to be some of the first diagnostic indicators of pathology and initiators of the dentinal defense mechanism [7]. In animal patients the pain response is not as useful due to the fact that responses may be challenging to elicit. In order to understand the pain response of the tooth and its defense mechanism, a fundamental knowledge of the dentinal tubules and pulp is required. There are approximately 30 000–40 000 dentinal tubules per square millimeter of surface dentin [7]. In most domestic animals the odontoblastic process extends 0.2–1.5 mm into the dentinal tubule. In addition, there may be an afferent nerve fiber extending into the tubule 0.1–0.4 mm from the pulp. These fibers are sensory nociceptors, either A‐delta or C‐type fibers. A‐delta fibers are larger in diameter, are myelinated, and conduct nerve impulses more rapidly. They typically cause a rapid, sharp type of pain, often associated with a level of pulpitis that is still reversible. C‐type fibers are myelinated, have a smaller diameter, conduct impulses more slowly and elicit dull aches, as found in late pulpitis. As the response to thermal stimuli intensifies, the pain lasts well after the stimulus is removed. This inflamed pulp undergoes an irreversible change (irreversible pulpitis leading eventually to pulp necrosis). The tooth then will respond markedly to hot stimuli with cold stimuli potentially having a soothing effect on the tooth (an ice pack now relieves the pain). If a tooth responds to pain, it is said to have sensible dentin, which can provoke pulpal inflammation or pulpitis. While the presence of sensible dentin implies that a tooth is still vital, it does not necessarily indicate if the pulpitis is reversible or irreversible. Insensible dentin is generally indicative of a non‐vital tooth [8] In addition to the fibers, a dentinal tubule is filled with dentinal fluid, making it a hydrodynamic organ. Capillary permeability and blood pressure in the pulp results in intrapulpal pressures of 15–30 mmHg. The outwardly directed pressure gradient of the pulpal fluid gently drives it into the dentinal tubules, around the odontoblasts and nerve fibers, to become dentinal fluid. A slow outward flow of water and small molecules occurs through the dentinal tubules, even in areas covered with intact enamel or cementum, as these structures are permeable. If a dentinal tubule’s surface is exposed, the flowrate could be around 1 mm per hour [9]. A series of studies described by Brannstrom provided evidence that the main cause of dentinal pain is a rapid outward flow of fluid in the dentinal tubules that is initiated by strong capillary forces [10]. Cold stimulus caused a rapid outward flow of fluid at the pulpal end of the dentinal tubule, whereas heat stimulus caused a rapid inward flow of fluid. Rapid capillary action can also be caused by surface dehydration, friction (venturi effect), or fluid contraction. This fluid flow, although being insufficient to displace odontoblastic processes within the dentinal tubules, was sufficient enough to stimulate the sensory nerve endings in the underlying pulp dentin border zone [10]. Within the pulp are distinct cell zones, each with a specific function in healing. The cell layer closest to the tubules is the odontoblastic cell layer or primary cell layer. Next, there is a cell free zone, followed by a subodontoblastic cell‐rich zone or secondary cell zone (layer of Höhl) [7]. The primary layer of odontoblasts is a highly differentiated group of sensitive cells. These cells can be easily killed by toxins defusing through open tubules or from aspiration during rapid capillary action. A new generation of odontoblasts can come from the subodontoblastic cells, fibroblasts, undifferentiated mesenchymal cells from the pulp core, and vascular‐derived pericytes. These cells are more tolerant of toxins and can traverse the cell‐free zone to differentiate into odontoblasts. These newly differentiated cells can lay down new layers of reparative (tertiary) dentin to block the apertures of the dentinal tubules [11]. Pulpal inflammation, in association with sensible dentin, is the early defense mechanism for the tooth’s endodontic system [12]. In slight pulpitis, symptoms may not be apparent, but as the reactions increase, the healing sequence may be stimulated. Severe pulpitis may develop when profuse amounts of toxic products reach the pulp, accompanied by an excessive immunologic reaction, ultimately leading to pulpal necrosis [7]. Additionally, if the inflammatory or immune response is too weak or absent, the subsequent infection may result in liquefaction necrosis and pulpal death [12]. In mature teeth the pulp cavity is more narrow with a limited blood supply, with few, if any, undifferentiated mesenchymal cells left in a depleted cell‐rich zone of the pulp. These can limit the pulp’s ability to respond to disease [7]. When tubules are exposed on the surface due to injury, disease, abrasion, attrition, scaling, or root planing, material may accumulate at the surface aperture and eventually mineralize [8]. Materials such as dentinal fluid, salivary substances, fluorides, some lithotropic bacteria, and other substances accumulate at the aperture and then mineralize in a fashion similar to that of plaque mineralizing into calculus [7, 13]. However, in some cases, continued attrition or abrasion may prevent the tubules from being protectively sealed in this fashion. In these cases, removal of the continued source of wear and some form of dentinal sealer can be used to rectify the problem, or more advanced restorative procedures performed (inlays, onlays, crowns, etc.). Sclerotic dentin is more highly mineralized, with tubules being obliterated as they are filled with additional mineralization [7]. This process is similar to the surface mineralization, but extends well down into the tubule. For this to occur the odontoblast must have been lost, leaving a dead tract (unoccupied tubule). Tertiary (reparative or irregular secondary) dentin then forms at the tubule access, after which the sclerotic dentin can form [12]. Sclerotic dentin can also form in pulpless non‐vital teeth, although it may take more time [7]. It is seen in many older patients on exposed root surfaces as a highly translucent root dentin. Clinically, it is more difficult to bond a composite resin restoration to this type of dentin. Tertiary dentin is of two types, either reactionary, where dentin is formed from a pre‐existing odontoblast, or reparative, where newly differentiated odontoblast‐like cells are formed due to the death of the original odontoblasts, from a pulpal progenitor cell [11]. Tertiary dentin is deposited rapidly, often with a sparse and irregular tubular pattern. It sometimes contains cellular inclusions within its structure (osteodentin). This provides a positive effect to seal the pulp cavity from invasion by toxins and microbes. However, it should also be realized that reparative dentin can also have negative effects [14]. To begin with, it can result in a response that causes no pain, therefore not stimulating the endodontic immune system to mount a response to impending disease. Second, it can result in a reduced pulp canal, which can cause problems when accessing, or in instrumentation of the pulp chamber and canal when performing endodontic procedures. When a defect occurs in the hard tissues of the tooth (enamel, dentin), optimally it is best to preserve the function and structure of the object by restorative means. It is essential to know basic components of restorative efforts before undertaking therapy, including knowledge of cavity preparation, from skills involved to the final preparation that is required. These rules include conservation, esthetics, contours and contacts, cavity preparation, and identification with resolution of the cause. The conservation of natural tooth structure is the first rule of operative dentistry [3]. Conservation of tooth structure is essential for protection of the vital pulp. However, not only must the depth of preparationbe considered but also the size of the area. There are 30 000–40 000 odontoblasts per millimeter of dentinal surface area [7]. Therefore, a one‐centimeter squared preparation into the dentin will injure between three and four million odontoblasts in the pulp cavity, by severing off some degree of their processes or Tomes fibers. The degree of injury determines whether the individual odontoblasts become non‐vital. Crown preparation for a full coverage on a vital tooth will injure and irritate all of the odontoblasts in the crown pulp chamber. The more odontoblasts irritated, the more the pulp will be irritated. Natural, healthy, unmarred enamel that is supported by health dentin, pulp, and periodontium is the most esthetically pleasing. Therefore, the conservation of these healthy tissues is the best esthetic outcome possible. However, when tooth structure fails, esthetic restorative procedures come into play. The functional and esthetic outcome desired by the owner will then dictate the design of the operative preparation. A good general knowledge of dental anatomy is required to understand the physiology and function of tooth crown contours (see Chapter 1 – Oral Anatomy and Physiology). Contact areas, marginal form, and the buccal and lingual contours must be properly designed to reduce food impaction during mastication. Typically, contact areas should be restored to the condition that was present when the tooth was young and healthy. Restoration of the axial contours (buccal bulge, etc.) should be performed to protect periodontal health. See later in the chapter. If the cause of the disease can be ascertained, steps should be taken to relieve it and prevent it from recurring. Long‐term success of any restoration will be in doubt if the cause cannot be identified and resolved. This is important when dealing with pets that have habits such as chewing on cages, rocks, or sticks. In police, military, and protection trained dogs the source of the trauma can usually be identified, but not removed. In the required continuing bite training, the reinforced bite sleeve can sometimes cause damage, as well as actual on‐duty activity. If the bite sleeve caused an injury, it should be examined to determine if it can be modified or improved to reduce the probability of a recurrence of the injury. Dogs with a strong grip and rotational apprehension methods can break the teeth subgingivally. Abrasion patterns of incisors can be seen with atopic dogs, so dermatologic interventions may be needed. Any object that is non‐compressive or non‐bendable can cause tooth fractures with heavy chewing, especially of the carnassial teeth. Items with cloth or fibrous coverings can be extremely abrasive, especially if allowed to collect dirt or sand on surfaces. Play (or serious) fighting with other dogs or games of tug‐of‐war can also cause injuries to incisors and canine teeth Treatment planning requires a systematic approach to assess the structures and associated problems that may challenge treatment success. A close study of existing conditions that have led to the problem is required. It should be determined if modification in behavior, environment, or a combination of both is required. Additionally, the patient’s occlusion and periodontal health must be taken into consideration to optimize success [3]. The tooth structure must be evaluated for the ability to sustain a load, its relative retentive qualities, and esthetic requirements. Various walls, lines, and angles are created during cavity preparation. The following terms are used to identify the various components of a cavity prepared for restoration. An enclosing side of a prepared cavity is termed a wall. The wall is named in relation to the tooth surface of which it is formed. There are two internal walls possible, the axial and pulpal walls. The axial wall is the internal wall formed by the surface of the long axis (axial or vertical plane) of the tooth. The pulpal wall is the internal wall in the horizontal plane (Figure 17.2). There are numerous non‐internal wall surface potentials in a cavity preparation. Some of these are: Additionally, there are a few subdivisions of the walls, such as the enamel and dentinal walls. The enamel wall is that portion of the preparation wall that consists of enamel. The dentinal wall is that portion of the wall that consists of dentin. The dentinoenamel junction is that juncture in the wall where the dentinal and enamel walls meet. Where two walls meet a line angle is formed. At the point where three walls meet a point angle is produced (Figure 17.3). The cavosurface angle is the line angle formed between a wall of the prepared surface and the unprepared tooth surface. The cavosurface angle is also sometimes termed the preparation margin, especially once the preparation is restored. The combined peripheral extent of all of the cavosurfaces or preparation margins is termed the cavity or preparation outline. In dealing with restoratives, the restorative margin is the restorative surface that abuts the cavosurface angle or preparation margin. Design of the cavosurface angle requires special consideration in its preparation. The preparation marginal restoration greatly affects retentive qualities of the restoration, resistance to marginal leakage, physiologic contour reactions, gingival health, and resistance to attrition, abrasion, and fracture of the restoration and restored tooth. Selection of the specific cavosurface angle treatment is dependent upon the type of restoration selected, restorative materials to be used, degree of anticipated stress demand upon the restoration, and the length and direction of the enamel prisms (Figure 17.4). The mechanical cavity preparation involves the removal of defective, injured, or infected enamel and/or dentin. Affected (not infected) dentin is preserved. The cavity is then filled with a suitable restorative material that will reestablish the health, function, and often the esthetics of the tooth as well as its contour and shape. Minimal intervention dentistry has taken over from G.V. Black’s old philosophy of extension for prevention. However, G.V. Black’s other ideas on cavity design and tooth preparation are still relevant today [15]. Today, minimal intervention dentistry or conservative cavity preparation is designed to preserve as much healthy tooth structure as possible, only limited by access to diseased tissues and dental material requirements. This philosophy is the goal of restorative clinicians around the world. Modern cavity preparation and design and the evolution thereof cannot, or perhaps should not, be considered without reference to G.V. Black. Black’s text A Work on Operative Dentistry in 1908 was the first to prescribe a systematic method of cavity preparation and the ‘ideal’ cavity form [15]. Multiple factors must be taken into consideration prior to the design of the preparation outline being implemented upon the tooth, including location, extent, stresses, tooth condition, and esthetics. First, the classification by location by G.V. Black or similar classification is required. This classification will then direct certain biological mechanical principles to be applied. Next, classification by extent is a necessity. This usually does not overtly affect the cavity outline, but more the depth of preparation. This in turn determines whether cavity liners, indirect pulp capping, direct pulp capping, or root canal procedures will be required. Third, the occlusal and leverage stresses that must be encountered must be carefully studied. This impacts the types of restorative materials used and whether occlusal height of the crown should be reduced to diminish occlusal and leverage stresses. Four, the general condition of the tooth, including the presence of other restoratives currently in place or to be placed, must be contemplated. Finally, the esthetic demands by the client will need assessment. For example, the use of porcelain fused to metal (PFM) for the crown will require greater tooth reduction than the use for a typical metal crown. A more recent classification of lesions and general principles are determined by the nature and extent of the lesion, the quantity and quality of the tooth tissue remaining following preparation, functional load, and the nature and properties of the restorative system to be used [16]. In general terms, the minimal tooth substance is removed to allow access to the diseased tissues as well as allowing space for the requirements of the restorative material. For the removal of dental caries, an understanding of caries progression, tooth anatomy, including the position of the pulp (often based on an intraoral radiograph) and dental material science, is essential before starting. Preparation of a tooth to accept any form of restorative material requires specific steps, requiring planning, instrumentation skills, and an attention to detail. Dr. G.V. Black, almost a 100 years ago, set forth the basic sequence of tooth preparation for restoration [17]. This sequence included: outline form, resistance form, retention form, convenience form, pathology removal form, wall form, and preparation cleansing form. In addition to these, marginal placement and pulpal protection are also closely observed. Outline form consists of the external and internal pattern boundaries of the preparation. This includes consideration of the area of pathology, all undermined enamel, and adjacent pathology, tooth contours, and anomalous anatomy. The preparation margins are placed in areas least susceptible to pathology, where visualization and finishing are suitable for the operator, and where access for warranted hygiene by the client is adequate. In addition, the outline form must take into consideration access to the pathology, type of restorative material used, functional needs of the patient, and esthetic requirements. It is generally necessary to gain access using a friction‐retained, water‐cooled, diamond bur held in an air turbine handpiece. Diamond burs cut enamel very efficiently and would be the bur of choice for accessing caries through the enamel. The original tooth contour should be reestablished where possible and the reproduction of the contact point, if present, is important in preventing food impaction problems, which can lead to periodontal disease. Resistance form is the shape formulated for the preparation to resist fracture of the tooth and restoration, both during insertion and function. This would encompass the functional needs of the restorative material selected, by adequate reduction preparation for the volume of restorative material required and the correct angulation form of the walls to withstand the functional forces of occlusion. Any preparation will weaken a tooth and predispose it to fracture. To minimize this effect, all internal line angles should be rounded. Any increase in cavity depth can lead to flexure of cusp walls, which may predispose them to fracture. Rounding or curving the floor of the preparation can assist in minimizing this flexure. If a cusp has been totally undermined with no supporting dentin, then the operator should consider cusp reduction and a cusp overlay with a restorative material to minimize cusp fracture. Retention form is the shaping of the internal aspects of the preparation to assist in the prevention of the displacement of the restoration. This includes retentive undercuts, groove cuts to prevent rotation, dovetails, pins, posts, and the internal wall angle form. Today, with the use of adhesive materials, the need for pins, grooves etc. are less warranted. However, if a substantial amount of tooth structure has been lost, then an indirect type of restoration may need to be considered. In summary, the choice of material will influence the final form of the preparation, especially with the need for undercuts for non‐adhesive restorations such as amalgam. Convenience form is the shaping of the preparation in order to provide adequate visualization, suitable accessibility, and reasonable ease in placement of the restoration and its finishing. Pathology removal form is the shaping of the preparation that is necessary to remove or compensate for diseased, injured, or esthetically unpleasing dental tissue. The extent of pathology removal many times determines the need for the use of materials or agents to protect vital pulps. This may also result in the need for specific endodontic procedures. In the case of caries removal, caries should be initially removed from around the amelodentinal junction and then, working apically, toward the areas overlying the pulp. If caries extends down to the pulp, the operator will need to make a decision on whether to leave affected dentin or slightly soft dentin behind, so as not to enter the pulp. The use of caries detecting solutions may or may not assist the operator in identifying infected versus affected dentin. Affected dentin can be remineralized with the use of a therapeutic liner. The area of the amelodentinal junction must always be made completely caries‐free. Wall form is the refinement in the shaping of the preparation. This is typically required to eliminate unsupported enamel rods at the margin, or the smoothing of an irregular or rough outline form. Cleansing form is typically the final shaping of the preparation prior to restoration placement. It is generally accomplished with explorers, air, water, spray, cotton pellets, and other agents to remove debris from the preparation. G.V. Black originally proposed that margins should be placed well into the embrasures in cleansable areas and sometimes subgingivally. It is now accepted that margins should be kept free of the gingivae where possible to avoid periodontal problems and that incidence of overhangs and marginal gaps must be avoided. Any encroachment of the biologic width (approximately 2.5 mm from the margin of restoration to the crestal bone) will lead to periodontal inflammation. If the margin does violate biologic width, then a crown lengthening procedure will need to be performed prior to placing the restoration. As younger patients tend to have larger pulp chambers than older patients, inadvertent pulp exposure during the restorative procedure is a risk in the younger patient. A diagnostic radiograph showing the size and position of the pulp is mandatory. Where the operator gets within 0.5 mm of the pulp, pulpal protection is required in the form of a pulp capping material such as calcium hydroxide or mineral trioxide aggregate (MTA). Deep cavities may also require the need for a liner or base prior to restorative placement (see Chapter 16 – Advanced Endodontic Therapy). During operative procedures, various isolation schemes are used to enhance visualization, instrumentation, control moisture contamination from instruments, reduce salivary interference, and protect the patient from instrument or chemical injury. The isolation may be either for single teeth or entire arches. There are many methods for isolation, but the type and extent of isolation selected is determined by the type of procedure, length of procedure, area anatomy, and operator requirements. Moisture contamination including blood or saliva can interfere with the bonding of unfilled resins (bonding agent) to tooth structure as well as the cohesive bond between the composite resin and the unfilled resin. Blood contamination can also stain or discolor the restoration. Saliva may also contaminate the site with bacteria, especially when performing endodontic therapy on a tooth. The mouth mirror in combination with suction can be a highly expedient tool for isolation of an area. The mirror can be used as a lip or soft tissue retractor, an indirect visual aid, and to redirect light to an area to improve visualization. Suction can provide moisture and debris control for a clear field of view. Cotton rolls are tubes of absorbent material used to help control moisture at a site. They may improve or hinder visualization and access when in place and come in an assortment of diameters and lengths. Cotton rolls and hybrid cotton can be used to help isolate teeth, absorb excess moisture, occlude salivary duct openings, to aid in cheek retraction, or to apply medicaments. Rolls are used for tooth isolation for restorations and topical treatments, such as fluoride. They are placed in the buccal or lingual vestibule to aid in the control of moisture, being replaced as they become saturated. In the lower arch, cotton rolls and holders are sometimes used to provide retraction for access and improved visualization. Lip retractors are useful when working on the maxillary teeth to help prevent moisture contamination when placing a restoration. An atraumatic retractor can lift the upper lip out of the way, especially when working on rostral teeth. Cheek retractors can be made of metal or plastic and be single or double ended. They are used primarily to displace the cheeks away from the caudal teeth, either for dental visualization or shielding of soft tissues in dental procedures or for visualization during photography. Most tongue retractors are made of metal with rubberized tongue grips, and tongue shields are typically made of plastic or metal. Tongue retractors are used to move the tongue out of the way for procedures, while shields or guards generally partially cover the tongue for its protection. A rubber dam is a thin sheet of rubber or latex used to isolate an operating field in the oral cavity. The rubber dam is by far the most effective method of tooth isolation and moisture control (Figure 17.5). However, it may be difficult to place in dogs and cats due to a lack of customized rubber dam clamps. It can provide an area that is easier to maintain asepsis, a dry field, retraction of soft and hard tissues during oral treatments, and also allows for better visualization of the operative field. Latex gloves with holes punched into them can be used as rubber dams in animals, as they can be easily slipped over the muzzle and hold their position more naturally, particularly when working on canine teeth. True rubber dams come in various thicknesses and sizes for use according to the size of the oral cavity and teeth. The heaviest thickness that can be managed for an area is generally best. Some form of stabilization is needed to maintain the placement, with rubber dam holders, clamps, ligatures, interproximal devices, and tooth attachments. Care should be taken to avoid damage to surrounding gingiva. These may be applied to control gingival hemorrhage. A retraction cord can be placed, especially when restoring Class V lesions at the gingival margin. The retraction cord comes in various thicknesses. It can be soaked in astringent such as ferric sulfate to assist in the control of bleeding. The retraction cord also slows down the flow of gingival crevicular fluid from the gingival sulcus. The infiltration of a local anesthetic agent with adrenaline may also aid in the control of bleeding. Detection of lesions, such as caries, TR, and enamel defects is commonly performed with a sharp explorer, mouth mirror, good lighting, air syringe, and intraoral radiographs. Early detection of enamel disease is most reliant upon visualization and tactile inspection of the teeth. Incipient carious lesions of enamel may appear as rough or chalky white in good light, when air is blown across it. Sharp explorers, when pressed into a dental disease lesion, will ordinarily stick or catch on withdrawal. In moderate to advanced carious lesions of enamel, a brown to black appearance may develop in the pit, fissure, or developmental grooves, but must be differentiated from staining. In moderate to advanced resorptive and carious lesions, lucent areas may be detected radiographically. Laser caries detectors using laser fluorescence are now being used in human dentistry to detect early occlusal caries where radiographs and probing are equivocal. Once lesions have been detected, both the extent of involvement must be ascertained, as well as the relationship to the pulp cavity and pulp vitality. Near‐pulpal exposures can typically be visually detected by the pink hue of the dentin. This will usually be an indication for an indirect pulp capping procedure (see Chapter 16 – Advanced Endodontic Therapy). If the pulp has been exposed, but is still vital, a direct pulp capping or possibly a complete root canal procedure may be warranted. If the canal is exposed and the pulp is non‐vital, or is expected to become so, then a complete root canal procedure prior to restoration would be the treatment of choice. Operative chairside restorations generally involve the use of one, or more, of three basic restorative materials: composite resins, GIC, and amalgam. These products are held in place by micromechanical retention, chemical crystal formations, or macromechanical retention. Micromechanical retention is obtained by the use of bonding agents that microscopically interlock in enamel porosities, dentinal tubules, or other microscopic anatomy. This is used primarily with light cured composites and bonded amalgam restorations. Chemical crystal formations occur with glass ionomers as they form a crystal between the ionomer and the minerals within the enamel and dentin. Macromechanical retention are undercuts in the dentin and are used with non‐bonded amalgams and self‐ or autocure composites. The potential for the use of acrylic resins for permanent restorations in dentistry first began to be realized in 1955, when Buonocore reported upon the use of phosphoric acid on the tooth surface. He found that etching enamel dramatically enhanced the bonding of acrylic to the surface [18]. In 1962, Bowen introduced the new resin we today call composite, a reaction product of bis‐phenol A and a glycidyl methacrylate, commonly abbreviated to bis‐GMA [19]. The original formula was marketed as a powder–liquid system and a paste–paste form, both of which were self‐ or chemical‐cured products. In 1972, the first light‐cured composite resins were developed, which used an ultraviolet 365 nm curing light source [20]. This resulted in a controlled working and setting time. Most clinically used composites today use the visible light range of 460–480 nm, which provides a more controlled curing in a clinical setting. Today’s composites can be bonded to enamel, dentin, cementum, metals, porcelain, glass ionomers, and of course to other composites [20]. The composite resins on the market today come as a chemical cure, visible light cure, and ultraviolet light cured. The ultraviolet light cured resins are used mostly for indirect techniques using dental laboratories, while the chemical and visible light cured products are used predominately in clinics. The chemically activated resins normally use benzoyl peroxide as an activator. With the light cured products, most ultraviolet systems use benzoin methyl ether, while visible light systems use camphoroquinone [20].
Restorative Dentistry
17.1 Introduction
17.2 Restorative Terminology
17.2.1 Surfaces of Teeth [2]
17.2.2 Classifications of Cavities and Restorations
Class I
I, PM, M
Beginning in structural defects, such as pit or fissure, commonly found on occlusal surfaces (occlusal lesions)
Class II
PM, M
Proximal surfaces; when a tooth with a class 2 lesion includes a class 1, it is still considered as class 2 (proximal surfaces posterior/caudal teeth)
Class III
I, C
Proximal surfaces, incisal angle not included (proximal surfaces anterior/rostral teeth)
Class IV
I, C
Proximal surfaces, incisal angle included (proximal surfaces anterior/rostral teeth involving the incisal angle)
Class V
I, C, PM, M
Facial or lingual, gingival third; excluding pit or fissure lesions (cervical surfaces)
Class VI
I, C, PM, M
Defect of incisal edge or cusp; not included in Black’s original classification.
17.2.2.1 Elementary Cavity Class [3]
17.2.3 Dental Fracture Class
17.2.4 Staging of Tooth Injuries [5, 6]
17.2.5 AVDC Dental Abbreviations – Restorative AVDC Nomenclature
AB
Abrasion
AT
Attrition
C
Caries
DP
Defect preparation (prior to filling a dental defect)
E
Enamel
E/D
Enamel defect
E/H
Enamel hypoplasia
E/HM
Enamel hypomineralization
PCB
Post‐and‐core buildup
PCD
Direct pulp capping
PCI
Indirect pulp capping
R
Restoration (filling of a dental defect)
R/A
Filling made of amalgam
R/C
Filling made of composite
R/CP
Filling made of compomer
R/I
Filling made of glass ionomer
T/FX
Tooth fracture
T/NE
Tooth near pulp exposure
T/PE
Tooth/pulp exposure
Other abbreviations that may be found:
DB
Dentin bonding agent
P&F
Pit and fissure
P&FS
Pit and fissure sealer
SI
Staining, intrinsic (blood, tetracycline, etc.)
SE
Staining, extrinsic (metal, food etc.)
VBL
Vital bleaching
NVBL
Non‐vital bleaching
VER
Veneer
17.3 Dental Defense Mechanisms
17.3.1 Successful Dental Defense Mechanism Sequence
17.3.1.1 Pain
,
though there can be areas of dentin in which there are no neural fibers in the tubules to elicit a pain response [9]. This can result in an insensible dentin response in a healthy vital tooth. It is interesting to note that while the afferent nerve endings respond to a variety of stimuli (temperature, pressure, etc.), the perceived sense is one of pain.
17.3.1.2 Pulpitis
17.3.1.3 Reparative Dentin
17.4 Basic Concepts of Restorative Procedures
17.4.1 Conservation of Natural Tooth Structure
17.4.2 Esthetics
17.4.3 Contours and Contacts
17.4.4 Cavity Preparation
17.4.5 Identification and Resolution of Cause
17.5 Treatment Planning
17.5.1 Components of Prepared Cavities
17.5.2 Preparation of Cavosurface Angles or Marginal Finish Lines
17.5.3 Modern Cavity Preparation
17.6 Steps of Cavity Preparation
17.6.1 Outline Form
17.6.2 Resistance Form
17.6.3 Retention Form
17.6.4 Convenience Form
17.6.5 Pathology Removal Form
17.6.6 Wall Form
17.6.7 Preparation Cleansing Form
17.6.8 Margin Placement
17.6.9 Pulpal Protection
17.7 Operating Fields
17.7.1 Mouth Mirror and Suction
17.7.2 Cotton Rolls
17.7.3 Retractors and Shields
17.7.4 Rubber Dam
17.7.5 Astringents and Retraction Cord
17.7.6 Lesion and Caries Detection
17.8 Restorative Materials
17.8.1 Composites