Elbow arthroscopy has been most commonly performed in medium to large breed dogs but has also been reported in small dogs (Hans et al. 2016) and in cats (Staiger and Beale 2005). Elbow joint arthroscopy is indicated when there is front leg lameness with elbow pain, crepitus, joint capsule distension, joint thickening or swelling, reduced range of joint motion, or when there are radiographic changes compatible with medial coronoid process disease (MCPD or FCP), osteochondritis dissecans (OCD), ununited anconeal process (UAP), intra‐articular fractures, or any visible degenerative changes. Significant MCPD can be present in the radiographically normal joint so even very subtle radiographic changes compatible with MCPD are a definite indication for arthroscopy. Pain on palpation of the craniomedial aspect of the elbow joint over the medial coronoid process, pain with full flexion of the elbow joint, or pain with internal or external rotation of the antebrachium is strongly suggestive of MCPD. Absence of pain does not rule out the disease. Localized swelling in the craniomedial aspect of the joint over the medial coronoid process can also be helpful in establishing an indication for arthroscopy. Crepitus is not commonly detected with MCPD and is more likely to be found when there is UAP. Joint capsule distension or joint capsule thickening is nonspecific and can be seen with any of the disease conditions that occur in the elbow joint but is a clear indication for arthroscopy. Generalized swelling or thickening of the joint is also nonspecific, is an indication for arthroscopy, and typically is suggestive of severe joint disease, especially if combined with reduced range of joint motion. Joints with reduced range of motion and swelling may require a more aggressive multiport approach to the joint for treatment of the originating pathology and for removal of multiple secondary osteophytes.
It is not important to differentiate between MCPD and OCD of the elbow joint before arthroscopy as patient positioning and portal placement are the same for both conditions. Unless radiographs are normal, CT or MRI is not needed to establish an indication for arthroscopy. CT is recommended before arthroscopy to improve definition of the pathology that is present and is especially important if there is severe joint pathology with multiple large osteophytes. The CT findings are used to help define the location of osteophytes or arthroliths that need to be removed. Information gained from a CT is very beneficial in planning the arthroscopic procedure.
When fragmented medial coronoid process first entered our index of suspicion when evaluating front leg lameness with radiographic changes in the elbow open surgical exploration was used as a diagnostic technique. Arthroscopy was first used by the author as a diagnostic technique to avoid open surgery just to make a diagnosis. Arthroscopy has contributed significantly to our knowledge of the multitude of changes seen with elbow dysplasia. With time and experience, arthroscopy became part of our treatment protocol. Arthroscopy has replaced open surgery as the procedure of choice for diagnosis and management of all forms of elbow dysplasia and provides superior results to other options (Barthélémy et al. 2014; Evans et al. 2008; Galindo‐Zamora et al. 2014). Although there is disagreement with this conclusion there is too much variation in results, case selection, experience, small numbers, and evaluation methods to indicate that arthroscopy is not indicated (Burton et al. 2011; Dempsey et al. 2019).
4.1 Patient Preparation, Positioning, and Operating Room Setup
The patient is typically placed in dorsal recumbency for elbow arthroscopy whether for bilateral or unilateral procedures. For bilateral procedures, the legs are clipped, suspended (Figure 2.2a and b), prepared, and draped to allow free manipulation of the leg and access to all sides of the elbow joint or joints. For unilateral procedures, the contralateral limb is retracted caudally out of the way and is tied to the surgery table. For unilateral MCPD, OCD, UAP, and multiport joint debridement procedures, two monitors are recommended, one at each end of the table (Figure 2.5), or the monitor can be placed across the table from the surgeon with the assistant standing on the cranial side of the surgeon (Figure 2.6). The monitor is placed at the head of the table for bilateral MCPD and OCD procedures with the assistant and surgeon on the same side of the table with the assistant standing on the cranial side of the surgeon between the monitor and the surgeon (Figure 2.4). Arthroscopy for bilateral UAP can be done with one monitor at the foot of the table (Figure 2.3) but is best done with two monitors, one at the head of the table and one at the foot of the table (Figure 2.5). MCPD is commonly associated with UAP and evaluation of the medial coronoid process with appropriate treatment is an important portion of arthroscopic management for UAP. The reported combination of these two pathologies is 16% (Meyer‐Lindenberg and Fehr 2006) but the authors’ experience suggests that the incidence of this combination is closer to 100%. An operative procedure in the craniomedial portion of the joint and in the caudal joint compartment are required for UAP management so without two monitors one of the procedures is performed working with the telescope pointing away from the monitor. This dramatically increases the difficulty of the procedure. Alternatives are to move the monitor between the different parts of the procedure or to perform unilateral procedures with the monitor placed across the table from the surgeon. Complete joint debridement with removal of multiple osteophytes through multiple telescope and operative portals are performed as unilateral procedures with the monitor placed across the table, at the head of the table, or preferably with two monitors. Use of two monitors allows the patient to be rolled from side to side and allows access to the medial aspect of the joint, the lateral aspect of the joint and for placement of caudal compartment portals.
4.2 Portal Sites and Portal Placement
4.2.1 Telescope Portals (Medial, Craniolateral, Caudomedial, and Caudal)
The most common telescope portal for the elbow joint is the medial portal (Figure 4.1) (Jardel et al. 2010; Tatarunas and Matera 2006; Van Ryssen et al. 1993). This portal is located directly distal or distal and caudal to the tip of the medial epicondyle of the humerus. The difference in this description does not change the portal position but the difference is based on the landmarks used to define portal location. If the shaft of the humerus is used for alignment, the portal is directly distal to the tip of the epicondyle but if the outside contour of the limb is used for alignment the portal is distal and caudal to the tip of the epicondyle. Descriptions of the location of this portal using the outside of the limb commonly use measured distances, like 1–1.5 cm, caudal to the epicondyle but variability in dog size makes this unhelpful. Comparison of palpation with distance based on radiographs of the patient can be used if palpation by itself is not enough to locate the correct site for portal placement. This portal site is located by palpating the tip of the epicondyle then sliding distally, in alignment with the humoral shaft, until the distal (caudal) margin of the superficial digital flexor muscle is palpated. Telescope insertion is facilitated by internal rotation (pronation) and abduction of the antebrachium to open the medial aspect of the joint. In thin dogs, the medial margin of the articular surface of the semilunar notch can be palpated as the antebrachium is internally and externally rotated opening and closing the medial aspect of the joint. The ulnar nerve can be palpated on the medial aspect of the elbow joint immediately caudal to the telescope portal site (Figure 4.1). The nerve is palpated before starting portal placement. A 20‐gauge 1.5″ hypodermic needle is placed into the joint at the telescope portal site to confirm accurate placement, joint fluid is aspirated, and the joint is distended with sterile saline. The ulnar nerve is palpated again at this point to confirm that the portal is being placed at a safe location. The needle can be removed or left in place, a stab incision is made into the joint with a no. 11 scalpel blade aligned parallel to the muscle fibers, and the telescope cannula is placed into the joint using the blunt obturator.
A variation of the medial telescope portal, the caudomedial telescope portal places it caudal to the ulnar nerve (Figure 4.2). The same technique is used except that the location is at the caudal margin of the medial supracondylar ridge. The primary indication for this portal is for removing medial humeral condylar ridge OCD lesions. These lesions are typically directly under the normal medial telescope portal making maintaining a visual field difficult. This proximity to the lesion also increases the difficulty for manipulation of instrumentation to remove the fragments and to debride the bed of the lesion. This more caudally placed portal decreases the difficulty of this procedure.
The craniolateral telescope portal (Figure 4.3) was the original telescope portal used by the author for the elbow joint but has been replaced by the medial portal and is no longer commonly used. Primary indications for this portal are for access to the elbow joint when the patient is in lateral recumbency with the joint to be examined on the uppermost side, for complete debridement of the elbow joint using multiple portals, for removal of coronoid process fragments that escape into the cranial compartment, and for arthroscopy‐assisted lateral humeral condyle fracture repair. This portal is at the intersection of the cranial margin of the radial head and the cranial aspect of the capitulum. The notch produced by this intersection can be palpated before joint capsule distension. When the joint is distended the joint capsule protrudes at this point to make a small bump and the telescope is inserted through this joint capsule prominence. If there is adequate joint distension due to the disease process, a 20‐gauge 1″ hypodermic needle is inserted at the portal site, joint fluid is aspirated, and the joint is distended with saline. A stab incision is made with a no. 11 scalpel blade and the telescope cannula is inserted using the blunt obturator. If there is inadequate joint distension secondary to elbow disease, a 20‐gauge 1.5″–2″ hypodermic needle is placed into the caudal joint compartment and the joint is distended with saline to allow portal placement. This portal can be difficult to place because there is a tendency for the blunt obturator of the telescope cannula to slip off the joint capsule and slide across the cranial aspect of the humeral condyle without entering the joint. This can be corrected by extending the stab incision through the joint capsule or using the sharp trocar for joint entry. Structures of the cranial compartment of the joint, including the medial coronoid process, can be evaluated with this portal by passing the telescope medially across the cranial aspect of the humoral condyle (Figure 4.4).
A lateral operative portal can be placed directly over the lateral coronoid process of the ulna. The caudolateral portal is commonly used as an egress portal but the same site is used for caudolateral operative or telescope portals. This caudolateral portal is placed caudal to the caudal margin of the supracondylar ridge of the humerus and proximal to the olecranon into the olecranon fossa.
Caudal telescope portals are placed into the caudal joint compartment by insertion into the olecranon fossa either medial or lateral to the triceps tendon. These portals allow visualization of the anconeal process and the olecranon fossa. The caudal portal on the lateral side can also provide access to the lateral coronoid process. Primary application of these portals is for removal of anconeal process osteophytes that interfere with joint extension as part of complete multiport joint debridement. The caudal portal on the lateral side can also be used when combined with a lateral operative portal for removal of lateral coronoid process pathology that cannot be accessed using the medial telescope portal. UAP fragment removal is generally performed using the medial telescope portal with a caudal operative portal on the medial side. Medial and lateral caudal portals can also be used for UAP removal and to evaluate the caudal compartment for any residual loose debris after the UAP fragment has been removed.
The craniomedial operative portal site can be used as a telescope portal site for visualization of radial head osteophytes, humeral condyle fractures, and MCPD fragments that have escaped into the cranial compartment of the joint. This site is used as a telescope portal site after it has been used as an operative portal site and the transfer can be done with a switching stick or the telescope cannula can be passed through the portal with the blunt obturator. Access to the cranial compartment of the joint through the standard medial telescope portal is facilitated by subtotal coronoidectomy.
4.2.2 Operative Portals (Craniomedial, Lateral, Craniolateral, and Caudal)
The craniomedial operative portal (Figures 4.1 and 4.2) is the most common operative portal for the elbow joint and is used for medial coronoid process disease management and for removal of OCD lesions of the medial humeral condylar ridge. This site is caudal to the medial collateral ligament and is located cranial and slightly proximal to the telescope portal. This site is directly over the medial coronoid process of the ulna providing excellent access and triangulation for fragment removal and coronoid process revision (Figure 4.5). The portal site is located with a 20‐gauge 1.0″ or 1.5″ needle, accurate placement is confirmed by visualizing the needle inside the joint with the telescope (Figure 4.6) (Video 2.2), a stab incision is made with a no. 11 scalpel blade parallel to the needle and aligned to pass between, not across, the muscle fibers (Figure 4.7), and a curved mosquito hemostat is used to dissect a portal tract into the joint (Figure 4.8) (Video 2.2). Operative cannulas are difficult to maintain at this portal site because of the small joint space and short distance of the bone from the joint capsule.
Access to the lateral coronoid process of the ulna and lateral ridge of the humeral condyle can be attained by entering the lateral aspect of the joint distal to the epicondyle using landmarks similar the medal telescope portal but on the lateral side. This operative portal can be combined with a craniolateral or caudolateral telescope portal or can also be used with a medial telescope portal for procedures requiring limited manipulations. Uses of this portal are for removal of lateral coronoid process pathology and for removal of medial coronoid process fragments or OCD fragments that escape into the lateral joint space. This portal can also be used as a telescope portal.
An operative portal can also be established at the craniolateral telescope portal site (Figure 4.3). This operative portal is used for removal of coronoid process fragments that escape into the cranial compartment of the joint and for removal of dorsal radial head osteophytes as part of a multiport elbow debridement procedure. This portal is usually placed from inside the joint using a craniomedial operative portal cannula and a switching stick.
Caudal compartment operative portals are placed either medial or lateral to the triceps tendon at the same locations as described for the caudal telescope portals. They are used for debridement of anconeal process osteophytes as part of multiport complete joint debridement combined with a caudal telescope portal. For this application, the operative portal is placed at the caudal portal site not used by the telescope. A caudal operative portal on the medial side is used for removal of UAP fragments combined with a medial telescope portal. The operative portal for UAP removal is more accurately a mini‐arthrotomy than a portal because it needs to be large enough for removal of the UAP fragment in one piece.
4.2.3 Egress Portals
The most common egress portal sites for the elbow joint are caudal portals on the medial or lateral sides with the egress cannula positioned into the olecranon fossa (Figures 4.1 and 4.2). The craniomedial operative portal site (Figures 4.1 and 4.2) can be used as an egress portal site during caudal joint compartment debridement or during UAP removal. For these procedures, the craniomedial site is used as an operative portal for managing medial coronoid process pathology and is then converted into an egress portal simply by placing an egress cannula into the already established operative portal site. Any of the elbow joint operative portal sites can also be used for outflow either with an egress cannula, with outflow around operative instruments, or through an operative cannula.
4.3 Nerves of Concern with Elbow Joint Arthroscopy
The ulnar nerve is within millimeters of the medial telescope portal of the elbow joint (Jardel et al. 2010). The ulnar nerve courses along the cranial border of the medial head of the triceps continuing on the medial aspect of the humerus immediately caudal to the point of the medial epicondyle to cross the medial aspect of the elbow joint. After crossing the elbow joint, the ulnar nerve courses distally on the caudomedial antebrachium between the humeral head of the superficial digital flexor muscle and the ulnar head of the flexor carpi ulnaris muscle (Figure 4.1). The nerve can be palpated as it crosses the medial epicondyle, allowing location of the nerve at the time of portal site selection and placement. Errors made in attempting to enter the elbow joint are to place the portal too far caudally directly over the nerve for the standard telescope portal or to be too far distally and to slide away from the joint on the medial surface of the ulna. The ulnar nerve is at risk when these errors occur.
The ulnar nerve is also within millimeters of the modified medial telescope portal of the elbow joint. This portal is placed caudal to the ulnar nerve distal to the epicondyle and caudal to the axis of the humeral shaft using palpation of the ulnar nerve as a landmark (Figure 4.2). The possible errors in placing this portal are reversed from the standard medial telescope portal with placement of this portal too far cranially or too far dorsally increasing the risk of ulnar nerve damage.
The median nerve is at risk with placement of the craniomedial operative portal of the elbow joint (Jardel et al. 2010). The median nerve crosses the medial extent of the flexor surface of the elbow joint, deep to the craniomedial flexor muscles, near the medial collateral ligament (Figure 4.1). This is close to the operative portal, within millimeters, but because the location of this portal is accurately established by intra‐articular observation of needle placement little risk is involved. Keeping this portal caudal to the medial collateral ligament, the standard position of the portal, minimizes the chance for damage to this nerve.
The craniolateral telescope portal is close to the radial nerve. The deep branch of the radial nerve crosses the flexor surface of the elbow joint, cranial and medial to this portal, and deep to the extensor muscles (Figure 4.3). Correct placement of this portal is into the protrusion of the distended craniolateral joint capsule at the junction of the articular surfaces of the capitulum and the radial head. There is little risk for radial nerve damage when the portal is properly placed at this location in adequate distension of the joint before portal placement or cranial displacement of the insertion site can cause the cannula to slide medially on the cranial surface of the joint capsule. The radial nerve is at risk when this occurs.
4.4 Examination Protocol and Normal Arthroscopic Anatomy
When first entering the elbow through the standard medial telescope portal, anatomic structures are identified that allow orientation within the joint. Maintaining the antebrachium in an internally rotated position with the medial aspect of the joint opened is essential for examination of the elbow joint. The medial coronoid process of the ulna, the radial head, the medial ridge of the humoral condyle, and the medial collateral ligament (Figure 4.9) are used for orientation. Other structures that can be used are the articulation between the ulna and the caudal margin of the radial head (Figure 4.10), the concave ridge of the semilunar notch with the convex surface of the humeral condyle, and the anconeal process (Figure 4.11). Once orientation is established, the joint is examined in a systematic manner to insure evaluation of all the important structures of the joint. Starting in the craniomedial portion of the joint with the telescope oriented cranially the medial coronoid process, medial aspect of the radial head, medial collateral ligament, and the medial ridge of the humeral condyle are evaluated (Figure 4.9). The telescope is swept caudolaterally to see the radial head (Figure 4.12) and continuing in this direction the lateral portion of the radial head articulating with the lateral ridge of the humeral condyle (Figure 4.13) and the radial notch of the ulna (Figure 4.14a). With application of additional rotational and abduction force, rotation of the telescope to face the 30° angle of view distally, and deeper insertion of the telescope, the lateral coronoid process can be seen in most elbows (Figure 4.14b). In this image, the caudal portion of the radial head, articulation of the radial head with lateral coronoid process, and the lateral ridge of the humeral condyle are also seen. There is extensive variation in the normal appearance of the lateral coronoid process from a thin projection with a narrow but well‐defined articulation with the humeral condyle (Figure 4.15) to wide and blunt with a large articular surface with the humeral condyle (Figure 4.16) or small and irregular with no articulation with the humeral condyle (Figure 4.17). Continued caudal angulation of the telescope exposes the semilunar notch and lateral ridge of the humeral condyle (Figure 4.18). The central portion of the semilunar notch with the base of the anconeal process and central portion of the articular surface of the trochlea of the humerus are seen when the telescope is pointed more caudally (Figure 4.19). When the telescope is pointed caudally, the anconeal process is seen with the caudal trochlea of the humeral condyle (Figure 4.20). With flexion of the joint, the tip of the anconeal process is visible (Figure 4.21) and in some joints, the telescope can be passed into the caudal compartment of the joint from the medial telescope portal (Figure 4.22).
Orientation when using the craniolateral telescope portal uses the cranial or dorsal aspect of the radial head (Figure 4.23), cranial articular surface of the humeral condyle (Figures 4.23 and 4.24), the cranial tip of the medial coronoid process (Figures 4.24 and 4.25), and attachment of joint capsule to the distal humerus (Figure 4.26). The cranial compartment of the joint can also be accessed for examination through the medial telescope portal after removal of the medial coronoid process or through a craniomedial operative portal. Access through the medial telescope portal is easier after a subtotal medial coronoidectomy has been performed. From the medial telescope portal, the arthroscope is passed through the space lateral to the medial collateral ligament (Figure 4.27). The dorsal aspect of the radial head and the cranial surface of the humeral condyle (Figure 4.28) are used for landmarks.
The caudal portals use the tip of the anconeal process and the caudal articular surfaces of the humeral trochlea for orientation (Figures 4.29 and 4.30). From the caudal portals, the soft tissue membrane of the supratrochlear foramen is seen (Figure 4.31). The telescope can be passed through the supratrochlear foramen (Figure 4.32) to visualize the cranial joint space (Figure 4.33).
Examination of the caudomedial joint space medial to the epiphysis of the humerus is needed to evaluate for injury of the origin of the extensor muscles of the carpus. This area is examined from the medial telescope portal by directing the telescope tip caudally and medially to enter the joint space medial to the bone and deep to the muscles. The reflection of the joint capsule at the proximal extent of the joint space with the tendon origin of the flexor muscles (Figure 4.34) and continuation of the tendon across the caudal margin of the supratrochlear ridge (Figure 4.35) are seen in this joint space.
The pars nudosa is a normal area without articular cartilage on the ulnar articular surface. This cartilage defect has a wide range of appearance and size including small shallow areas containing fat (Figure 4.36), deeper areas lined with periosteum (Figure 4.37), and large irregular areas (Figure 4.38).
The ligament of the radial head extends from its attachment on the base of the medial surface of the medial coronoid process around the radial head to attach to the lateral surface of the proximal ulna. The medial portion of this ligament can be seen medial and cranial to the medial collateral ligament with the telescope directed cranially and medially from the medial telescope portal (Figure 4.39).
Intra‐articular fat is a common finding and is seen in many areas of the elbow joint (Figures 4.6, 4.9, 4.22, 4.27, 4.31–4.33, and 4.36).
4.5 Diseases of the Elbow Diagnosed and Managed with Arthroscopy
4.5.1 Elbow Dysplasia
This entity has gone through a series of names over the past 30 years in an attempt to more accurately refer to a combination of abnormalities of the elbow joint. First called elbow dysplasia, then fragmented coronoid process (FCP), elbow incongruity, medial coronoid process disease (MCPD), medial coronoid process pathology (MCPP), and medial compartment syndrome (MCS). None of these names are any better suited than the others and none accurately describe the variation of underlying pathology. The first name, elbow dysplasia, is as good as any of the more recent monikers and more accurately describes our lack of understanding of this elbow joint pathology. The term elbow dysplasia when first used included ununited anconeal process and any degenerative changes seen on radiographs without other specific diagnoses, as they had not been defined at that time. With the addition of diagnoses including OCD, abnormal formation of the medial coronoid process, and abnormal interaction of the ulna with the radial head and humeral condyle, the term elbow dysplasia encompasses this spectrum of diseases but with more variation allowed.
Many theories have been discussed and published concerning the underlying etiology of elbow dysplasia. The more that is said, the more that is published, and the more elbow cases that are done the less is understood about the elbow. None of the current theories seem to cover all the changes that are seen. There are many interacting factors that come from multiple abnormalities that do not yet have answers. The need to keep looking and continue to question all the presented theories is definitely still present and will continue until we have a complete understanding of this disease.
In addition to pathology associated with interaction of the humeral condyle with the radial head and ulnar articular surface, there may also be pathology involving articulation of the articular circumference of the radial head with the radial notch of the ulna. If these two surfaces are not congruent then abnormal forces occur with subsequent fragmentation of the axial or lateral aspect of the medial coronoid process. Many medial coronoid process fragments fit this concept as an etiology. But this is not the only etiology.
I do not have the answers. Most of the theories presented look only at the adult elbow joint. The relationships of the involved bones during growth have not been studied and consideration needs to be given to possible changes during growth. In thinking about the changes seen in some elbow joints, it is evident that the relationship of radial and ulnar length, specifically the relationship of the weight‐bearing surfaces of the radial head to the semilunar notch, may not be the same during all phases of growth as it is in the adult dog. This could account for changes in both the coronoid process and the anconeal process. This could also account for an abnormal medial coronoid process with cartilage loss that sits below the level of the radial head. Or is the pathology in the humeral condyle from abnormal circumference or varus–valgus angulation. The remaining questions far overshadow the current answers.
A complete discussion of the history, theories, and pathophysiology of elbow dysplasia is beyond the scope of this work. The focus of this book is aimed at the applications, procedures, and techniques of arthroscopy. Those interested in gaining further knowledge on the abnormalities of the elbow need to pursue this in other publications. The comments in the previous paragraphs are the opinions of the author developed with 37 years of experience performing elbow arthroscopy, reading many of the publications on elbow disease, independent discussions with other surgeons, and group discussions with other surgeons at meetings.
Elbow dysplasia is the most common indication for, and diagnosis achieved with arthroscopy of the elbow joint. Arthroscopy of the elbow joint is indicated with front leg lameness in medium to large breed dogs when there is elbow joint pain, swelling, thickening, reduced range of motion, or there are any radiographic changes present in the elbow joint. Medial coronoid process changes are diagnosed primarily in young dogs but have been seen as an acute onset lameness in dogs as old as nine years. Definitive differentiation of the etiology of radiographic change is not necessary before arthroscopy as arthroscopy will allow determination of the diagnosis. The most common conditions seen in the elbow joint, medial coronoid process disease, ununited anconeal process, and OCD, are approached through the same medial telescope portal. Elbow CT is a very important addition to understanding the changes in individual elbows (Botazzoli et al. 2008; Coppieters et al. 2016a, b; Eljack and Böttcher 2015; Griffon et al. 2018; Groth et al. 2009; Kramer et al. 2006; Krotscheck et al. 2014; Lau et al. 2015; Moores et al. 2008; Skinner et al. 2015; Villamonte‐Chevalier et al. 2015; Wagner et al. 2007) but is not needed for arthroscopy to be indicated and performed. Elbow dysplasia is commonly a bilateral disease process and bilateral elbow arthroscopy is routinely recommended even in the presence of unilateral presentation. Unilateral presentation does not necessarily indicate unilateral involvement but may only indicate that the presenting elbow is more painful than the nonclinical elbow. In many cases, it seems that more benefit is achieved with arthroscopy for the asymptomatic joint than for the joint of presentation because it is treated at an earlier stage with less joint damage. It is also far easier for the patient and more economical for the client to perform a bilateral procedure than two independent unilateral procedures.
There are many open surgical procedures applied to the elbow joint for management of elbow dysplasia. Various forms of elbow osteotomy with or without fixation are the most common of these procedures. The primary humeral procedure is sliding humeral osteotomy. Open intraarticular procedures are also included in this list. It is the authors’ opinion that there is no science that supports any of these open surgical procedures providing any better long‐term results than arthroscopy by itself. These open surgeries are unnecessarily invasive, traumatic, and painful. They also require prolonged recovery and induce the risk for failure of the osteotomy to heal. For these reasons, it is the authors’ opinion that these open surgeries are not indicated. In line with Noel Fitzpatrick’s statement in an elbow dysplasia session at a recent ACVS Symposium, “There is nothing we can do to prevent progression of degenerative joint disease of the elbow joint.” If we believe in this statement and it probably is true, then all we are doing is pain management. The list of open surgeries, especially those involving osteotomies, cause pain for a significant period of time and there is no science to support that long‐term pain is reduced more than with arthroscopy alone.
Arthroscopy for elbow dysplasia is performed through a medial telescope portal and a craniomedial operative portal (Figure 4.1). An additional caudomedial operative portal is required when there is an ununited anconeal process and is at the site shown for the egress portal (Figure 4.1). Egress is typically through the operative portal. An egress portal can be placed in the caudal compartment of the joint if needed but is seldom required. Medial coronoid process disease is typically easily visible, but some lesions are subtle and are not easily seen on initial examination of the joint. There is an extensive variety of pathology that can be present with a wide presentation of lesion severity. The wide variation of lesions that are seen also indicates that this is not a single disease process but is a variety of different abnormalities producing a wide range of distinctly different lesions.
There is a wide range in size of free medial coronoid process fragments from small (Figure 4.40), to medium (Figure 4.41a and b), large (Figure 4.42), massive (Figure 4.43), and there can be multiple fragments of various sizes (Figures 4.44 and 4.45). Small coronoid process fragments with no other pathology are seen in only a small percentage of cases (Video 4.1). In these cases, normal cartilage is present on the free fragment, the fixed portion of the medial coronoid process, and on the medial ridge of the humeral condyle (Figure 4.46). Small free fragments are also seen with all grades of cartilage damage (Video 4.2). Classic larger free coronoid process fragments (Figure 4.42) and multiple fragments (Figure 4.47) are also occasionally seen with normal cartilage surfaces (Videos 4.3 and 4.4). Medial coronoid process fragments are most commonly free or loose movable bone as shown in the previous figures, but “fixed” fragments are not uncommon (Video 4.5). Many fragments are obviously loose, but the status of other fragments is only determined by palpation for movement (Video 4.6). Fixed fragments have the same range of size from small (Figure 4.48), medium (Figure 4.49), to large (Figure 4.50), and multiple (Figure 4.51) as do free fragments. Free and fixed fragments can be present in the same joint (Figure 4.52) (Video 4.7) or fixed fragments present in one joint (Figure 4.47) with a free fragment in the contralateral joint (Figure 4.53). The margins of fixed fragments can be clearly seen in some patients as a linear cartilage defect with no cartilage damage other than this linear defect (Figures 4.48–4.51), as indistinct linear chondromalacia (Figure 4.54), or as distinct linear cartilage mineralization (Figure 4.55). In other cases, there may be no visible margin with overlying normal cartilage (Figure 4.56), there may be no visible margin because the margin is obscured by chondromalacia (Figures 4.57 and 4.58), or the margin can be seen when there is Grade V chondromalacia and a fissure line is visible in the exposed bone (Figures 4.59 and 4.60). Fixed fragments that are hidden by normal cartilage or chondromalacia are best diagnosed with preoperative CT studies that define fissure lines. The hidden fissure lines can also be found by removal of the overlying cartilage (Figure 4.61).
Cartilage pathology is not related to free fragment size as small lesions are seen with extensive cartilage loss (Figure 4.40) and large fragments occur with minimal or no cartilage damage (Figure 4.50). Medial coronoid process fragments most commonly arise from the lateral side of the medial coronoid process occurring at any position along that margin, cranially (Figures 4.41, 4.46, 4.53), in the middle (Figures 4.40, 4.48–4.50), at the base (Figure 4.44), or can be the entire lateral margin of the process (Figures 4.42, 4.43, 4.45, 4.47, 4.51, 4.52). Fragments can also arise from other parts of the medial coronoid process including the apex (Figure 4.62) and uncommonly the medial margin (Figure 4.63).
Chondromalacia involving the medial coronoid process can be minimal with low‐grade minor changes involving small areas of the joint surface to full thickness cartilage loss with eburnation of exposed bone over an extensive area of the ulnar articular surface. Table 3.1 defines the modified Outerbridge chondromalacia grading system (Griffon 2012; Outerbridge 1961) used in these descriptions. Medial coronoid process disease is uncommonly seen with normal cartilage on all structures within the joint (Figures 4.41, 4.46–4.48, 4.50, and 4.51). More commonly there is damage to cartilage on the fixed portion of the medial coronoid process with the grade of chondromalacia seen on free medial coronoid process fragments typically being much less than that seen on the fixed portion of the medial coronoid process (Figures 4.40–4.45). Small Grade I lesions on the fixed portion of the medial coronoid process are seen showing blistering (Figure 4.64), softening (Figure 4.65), or swelling (Figure 4.56). Grade II lesions are seen as small areas with fibrillation (Figure 4.66), fissures (Figure 4.67), or loss of cartilage thickness (Figure 4.68). Larger lesions with deeper cartilage involvement represent Grade III chondromalacia with fibrillation (Figure 4.58), fissures, uniform loss of cartilage thickness (Figure 4.69), or loss of cartilage thickness with a moth‐eaten appearance (Figure 4.57). Grade IV chondromalacia signifies full thickness cartilage damage with exposed bone (Figure 4.70) seen as uniform loss of cartilage thickness (Figure 4.45) or irregular moth‐eaten cartilage loss (Figure 4.71). Grade V chondromalacia is seen on the fixed portion of the medial coronoid process as complete loss of cartilage and smooth exposed and eburnated bone with feathered cartilage margins (Figures 4.40, 4.44, and 4.60).
Chondromalacia also commonly occurs on the humoral condyle with all Grades being seen. Minor or Grade I chondromalacia appears as swelling (Figure 4.72), blisters (Figure 4.73), or softening (Figure 4.74). Small partial thickness, Grade II chondromalacia, including erosions (Figure 4.75), partial thickness wear lesions (Figure 4.76), fibrillation (Figure 4.77), or moth‐eaten cartilage (Figure 4.78). Grade III chondromalacia lesions have the same range of appearance as Grade II lesions but are larger and deeper but do not penetrate to bone. Erosions (Figure 4.79), wear lesions as single grooves (Figure 4.80) or widespread cartilage damage (Figure 4.81), cartilage fibrillation (Figures 4.80 and 4.82), moth‐eaten cartilage (Figure 4.83), and cartilage fissures (Figure 4.84) are all representative of Grade III chondromalacia. Progression to exposed bone is termed Grade IV chondromalacia and is also expressed as erosions (Figures 4.45 and 4.85), wear lesions (Figure 4.86), cartilage fibrillation (Figure 4.87), and moth‐eaten cartilage (Figure 4.88). Extensive full thickness lesions with eburnation of exposed bone are given the category of Grade V chondromalacia. This grade was added to the original four Outerbridge grades as part of the “Modified” grading system (Griffon 2012) and is specifically suited to categorizing elbow pathology. Full thickness wear lesions are commonly seen on the medial ridge of the humeral condyle (Figures 4.40, 4.43, 4.44, and 4.60) and chondromalacia occasionally extends laterally onto the trochlea of the humeral condyle as Grade II (Figure 4.89), Grade III (Figure 4.90), Grade IV (Figure 4.91), and Grade V (Figure 4.92) lesions.
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